Any discussion of how to treat autism is sure to be problematic. To be

Any discussion of how to treat autism is sure to be problematic. To begin with, autism, a neurological condition that impairs an individual’s ability to interact with others, is a wide-ranging spectrum disorder. Some high-level autistics hold jobs and have families. Others are almost incapable of communicating or otherwise interacting with the world. Between these extremes lie individuals with a variety of symptoms. Some can communicate, but have difficulty differentiating between a trivial comment and an important point. Others cannot stand to be touched, or might feel a gentle touch as hurtful. As there is no nationwide agreement on the classification of different levels of autism, a person considered to have high-level autism (sometimes called Asperger’s Syndrome) could move to a state where he or she is deemed neurotypical—that is, not autistic at all.

Not knowing what causes autism also complicates any consideration of its treatment, though genetic research has proven promising in this regard. A region of chromosome 1 has been associated with autism, as has a region of chromosome 3 that contains the GAT1 gene and the OXTR gene. The GAT1 gene is involved in transmitting messages between brain cells, and the OXTR gene appears to help the early brain develop.

Environmental factors, including viral infections and exposure to environmental chemicals, continue to be investigated as well. There has also been research into the possibility of a link between autism and certain vaccines. None of these possible causes has proven conclusive, but none has been ruled out, either.

Finally, many autistics reject the premise that they need to be “cured” at all. These people, who prefer the term “autistics” to “people with autism,” believe that eradicating their autism would wipe out something central to their personalities. Many are opposed to a treatment known as Applied Behavioral Analysis (ABA), arguing that this is a potentially traumatic process that only teaches autistics to imitate certain neurotypical behavior. Some in the autistic community are also critical of neuroleptic drugs, which can suppress certain behaviors, but which, the critics say, are overprescribed. With so much controversy regarding the nature of this complicated condition, it is unlikely that any agreement as to its proper treatment, or whether it should be treated at all, will be forthcoming.

1. The primary purpose of the passage is to _______.

(A) provide a summary of the range of treatments available for autism
(B) give a general description of the symptoms and possible causes of autism
(C) show that autistics are not in agreement as to how to treat their condition, or whether to treat it at all
(D) reconcile various opinions regarding the treatment of autism
(E) present an overview of the difficulties involved with treating autism



2. According to the passage, an autistic individual _______.

(A) might have developed the condition through exposure to a vaccine
(B) is symptomatically unlike an individual with Asperger’s Syndrome
(C) given neuroleptic drugs stands a better chance of controlling his or her symptoms than an individual treated with ABA
(D) who is able to communicate with some proficiency would prefer being called “autistic” not “a person with autism”
(E) is not able to properly perceive the sense of touch



3. It can be inferred that individuals who prefer to be called “autistics” rather than “people with autism” _______.

(A) are likely to have Asperger’s Syndrome
(B) are skeptical that altering someone’s GAT1 gene would have any meaningful effect on their autism
(C) would not be in favor of genetic manipulation as a way to eliminate their autism
(D) are able to communicate verbally with relative ease
(E) do not believe that autism is a crucial part of their personalities



4. According to the passage, brain development in infants _______.

(A) is largely determined by a region of chromosome 1
(B) can be affected by environmental chemicals
(C) is aided by the OXTR gene
(D) can be damaged by an overprescription of neuroleptic drugs
(E) can be altered by ABA